BEGIN:VCALENDAR
VERSION:2.0
PRODID:-//ZContent.net//ZapCalLib 1.0//EN
CALSCALE:GREGORIAN
METHOD:PUBLISH
BEGIN:VEVENT
SUMMARY:Plant disease resistance mechanisms: Two stories
LOCATION:Anatomy/Zoology Building W118
TZID:America/Denver
DTSTART:20162201T000000
UID:2026-10-01-06-14-14@natsci.colostate.edu
DTSTAMP:20261001T061414
Description:Although some widely used paradigms have been identified\, plan
 t disease resistance operates by a multitude of mechanisms.  We have been
  studying the unique mechanisms of resistance to the most damaging disease
  of soybean\, caused by soybean cyst nematode.  We found that the most us
 eful resistance to SCN\, which is present in many of the most successful s
 oybean varieties\, relies on an intriguing form of copy number variation i
 nvolving up to ten copies of a 31kb genome segment that encodes four dispa
 rate proteins.  Our most recent work on this Rhg1-mediated resistance wil
 l be discussed.  In a separate project we have found that diverse plant p
 athogens\, early in the infection process\, induce DNA double-strand break
 s in the host plant genome.  Plants require the capacity to limit or repa
 ir this DNA damage as part of an effective immune system\, and our more re
 cent work on these processes will be presented in the second part of the s
 eminar. 4:00 pm
END:VEVENT
END:VCALENDAR
